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The restoration of mTOR signaling with Fer1 rescue treatment in the context of RSL3 effects, and the genetic knockdown of GPX4, reaffirm our in vitro findings that RSL3-mediated induction of ferroptosis, mTOR pathway suppression, DNA damage and failure of DNA repair, and autophagy are GPX4-dependent processes in thyroid cancer cells

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doi: 10.1016/J.IJBIOMAC.2024.131257 43 DongQ.GuoX.LiL.YuC.NieL.TianW.et al

This phenomenon shows that persistent luminal Ca 2+ depletion circumvents synthesis of ferroptosis-sensitive substrates in human cancer cell lines