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Potential mechanisms involved in this inflammatory response are hypoxia and cell death in the expanding adipose tissue, activation of the inflammation- and stress-induced kinases IB kinase- (IKK) and JUN N-terminal kinase (JNK), which further stimulate inflammation and insulin resistance [62]

the total levels, from each smoking article, of nictotine, nicotine-free dry particulate matter (NFDPM), carbon monoxide (CO), water, and the total particulate matter (TPM) is also shown