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By modulating TRP availability, central KP flux, glutamatergic signalling, oxidative stress, HPA-axis activity, and neuroplasticity, the microbiome-immune-BBB axis emerges as a mechanistic bridge linking peripheral physiology to central mood regulation

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Additionally, leveraging advanced therapies like exosomes within the realm of functional medicine can further enhance patient outcomes by promoting effective recovery and regeneration

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Since NF-B-driven transcription is a key regulator of pro-inflammatory cytokine expression, this blockade may account for the observed reductions in TNF- and IL-6 in LPS-stimulated models