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A number of generally un-testable hypotheses have been advanced to explain loss of ascorbate biosynthesis capacity in primates, teleost fish and other scattered animal species, including avoidance of H 2 O 2 production by gulonolactone oxidase and replacement of ascorbate with uric acid as an antioxidant since the loss of gulonolactone oxidase is correlated with loss of uricase [69]

We confirmed increased p-CREB and p-ERK levels in TC1-6 cells upon high-level glutamine and alanine treatment (Supplementary Fig

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