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Zhang, and J

Furthermore, by promoting Nrf2 expression and activating HO-1, it significantly reduces ROS levels and MDA content in the kidneys of AKI rats, increases levels of GSH, GPX4, and other anti-ferroptosis proteins, markedly reduces the number of apoptotic cells in the kidneys and renal tubular epithelial cells of model rats, and promote the expression levels of pro-apoptotic proteins, thereby protecting the kidneys (181)

placebo challenge (Figure 4)

While both compounds interact with GLP-1 and GIP pathways, Retatrutide differs as a triple agonist involving glucagon receptor activity