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These findings suggest that alterations in the excitatory neurotransmission regulation, presumably increased cycling of the Gln-Glu circuit, may underlie the pathophysiology of ASD

Kawicka, A., et al

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Herein, we aimed at examining the hypothesis stating that the excitatory glutamatergic neurotransmissions were enhanced in the autistic brains in association with the astrocytic activation which is related to Gln synthesis and disrupted dopaminergic signals through D1Rs

Activation of glial cells leads to the release of inflammatory cytokines, including IL-1, IL-6, and TNF-