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The incidence of chromosomal aberrations in the bone marrow of SAMP-1 mice with accelerated aging appeared almost twice as high as in the other two models

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1 When IGF-1 binds to IGF-1R, the tyrosine kinase activates the phosphoinositide 3-kinase (PI3K)-AKT pathway and many integral proteins involved in cellular metabolism, apoptosis, cell adhesion, and angiogenesis are regulated within this pathway

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