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Ang II pretreatment resulted in high blood pressure, downregulation of SIRT7, and excessive ROS generation, lipid peroxidation and renal ferroptosis by decreasing the expression of GPX4, SLC7A11, and NRF2 [263]

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The pSDU1-I-Sce I plasmid in mutant cells was eliminated by spontaneous loss, as described earlier (Wu et al., 2.9

Short-term infusion studies utilising the GLP-1/GIP combination compared with single GLP-1 infusion have failed to show an advantage of the combination over GLP-1 alone [49, 53, 54]