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Satb2 deletion decreased the energy metabolism of colonic epithelial cells by down-regulating the transcription of PPARG In further exploring how Satb2 contributes to intestinal mucosal repair, we initially hypothesized that its increased expression might promote the proliferation of colonic epithelial cells during the repair process following irradiation

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We recorded excitatory and inhibitory postsynaptic currents (EPSCs and IPSCs) at holding potentials of 60 and 10 mV, respectively