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GSTP1 forms a complex with TRAF2 and thereby prevents the formation of TNF--induced TRAF2-ASK1 complex and therefore suppresses the activation of apoptosis signal-regulated kinase 1 (ASK1) [65]

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The pathogenesis and the pathophysiology of sepsis is very complicated and multifaceted: numerous biological mechanisms, such as infection, inflammation, oxidative stress, immune system dysregulation, blood coagulation, endothelium dysfunction, tissue damage, elevated cellular death and/or apoptosis are involved during sepsis at tissue, cellular and molecular levels causing a damaging systemic reaction [379]

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