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Microglial complement receptor 3 can induce ferroptosis via NADPH oxidase 2 (NOX2) (60), and other NADPH oxidases, including NOX1 (61) and NOX4 (62), are also key contributors to the ferroptotic process

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Yes, chelation therapy has been studied in clinical trials, including the TACT (Trial to Assess Chelation Therapy) for heart disease

A more detailed analysis of hepatic macrophages during APAP hepatotoxicity and recovery showed that there was a progressive loss of Kupffer cells during the injury phase with recovery during regeneration [103]

Duration follows the four to six week protocol outlined above