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The enhanced hepatic mitochondrial activity in the context of TKT deficiency has been shown to be the result of increased phospholipid synthesis, particularly increased synthesis of phosphatidylcholine which represents the major phospholipid in the mitochondrial membrane
44 Similarly, a prospective study observed an increased risk of hepatic fibrosis in diabetic patients, which was significantly mitigated by SGLT2 inhibitors
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Notably, cell migration is a key element in HF development
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