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Mechanistically, STAT6 mediates transcriptional repression of peroxisome proliferator-activated receptor and its downstream fatty acid oxidation-related genes, culminating in lipid accumulation within RTECsa metabolic perturbation that drives fibrotic progression (Youjing et al., 2023)

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About 25 years ago, practitioners found that people with these HLA types were over-represented in having mold reactions

Data from the United Kingdom indicate that nearly one-fifth of adolescents develop fibrotic progression by age 24, while the overall pediatric prevalence reaches 1 in 40 (22)

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