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Consistent with the non-glycaemic effects of glucagon to inhibit food intake, enhance lipid metabolism and accelerate energy expenditure [70], a single-molecule GLP-1/GCGR co-agonist improved body weight and glucose control in mice with DIO beyond that observed with GLP-1 receptor agonism alone [114, 115]

As in earlier trials evaluating a 2.4 mg weekly dose of semaglutide in people with obesity and Type 2 diabetes, results showed that participants on that dose lost significantly more body weight than those on the placebo an average of 10.4% of their starting weight, compared with 3.9%

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