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doi: 10.1002/jimd.12475 18

rFGF21 treatment alleviates mitochondrial damage and apoptosis after TBI in mice Moreover, our further investigations revealed that additional rFGF21 administration was associated with a reduction in mitochondrial morphology damage, ROS levels, MDA levels, as well as a restoration of mitochondrial respiratory chain complexes activities following TBI (Fig

This maintains physiological pulsatile release patterns and preserves negative feedback mechanisms that prevent excessive GH accumulation

The funding bodies played no role in the design of the study and collection, analysis, and interpretation of data and in writing the manuscript

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