It plays a key role in transporting long-chain fatty acids into the mitochondria where theyre broken down to produce adenosine triphosphate (ATP), the bodys main source of cellular energy
(1997) 273:G9207
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The pharmacological activation of mitophagy, particularly through pathways such as PINK1/Parkin and Nrf2 signaling, mitigates ferroptosis-induced cellular damage and slows the progression of IDD by preserving NPC viability and reducing oxidative stress (56,65)
