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Guided by the glucose metabolic cascade, we comprehensively discuss nano-enabled strategies to achieve antitumor efficacy through inhibiting glucose transportation, blocking glucose supply, accelerating glucose depletion, suppressing glycolytic enzymes, and concurrently inhibiting oxidative phosphorylation

This ensures high adherence and dose consistency but creates a significant logistical and psychological barrier

Mechanistically, by inhibiting mTOR activity, we induced transient metabolic and activation rest of CAR T cell products

The interplay of glucagon-like Peptide-1 receptor trafficking and Signalling in pancreatic Beta cells

The induction of FTL and FTH1 was confirmed by western blot (Extended Data Fig