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experimental models and clinical observations report androgen-driven increases in circulating neutrophil numbers and altered functional programs, including enhanced tissue recruitment and changes in bactericidal function [33, 34]

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Study found that increasing endogenous and exogenous intestinal SCFAs can stimulate intestinal endocrine cells to secrete GLP-1 and PYY by binding to GPR41 and GPR43 (Canfora et al., 2019)

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