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In parallel, clinical translation should incorporate mechanistic biomarkers and patient stratification to balance efficacy and safety, enabling the identification of subgroups most likely to benefit from ferroptosis-targeted interventions
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On the other hand, the plasma membrane glutathione efflux can be facilitated by specifically or ubiquitously expressed membrane proteins and anion channels such as multidrug resistance-associated proteins (MRP1-5), Cystic Fibrosis Transmembrane Conductance Regulator (CFTR), Arginine/Ornithine Transport ATP-binding Proteins (OATP 1,2), and ATP-Binding Cassette superfamily G member 2 (ABCG2) [19]
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