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DES(1-3) IGF-1 is a truncated variant missing the first three N-terminal amino acids (Gly-Pro-Glu), which also reduces IGFBP binding but through a different structural mechanism removal of the N-terminal tripeptide rather than charge substitution

mTOR inhibition with rapamycin (sirolimus) robustly induces autophagy, reducing tau and amyloid- aggregation in AD models as well as mutant huntingtin in HD models

doi: 10.1155/2012/510483