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Research also highlights possible effects on the nervous system

TRIM3 overexpression significantly improved the prognosis of patients with NSCLC in vivo

In pathological states such as AP, excessive Ca 2+ influx through Orai1 or mechanosensitive Piezo1 channels can overwhelm mitochondrial buffering capacity and promote mitochondrial dysfunction ( 2+ retention and exacerbating Ca 2+ imbalance ( 2+ but also indirectly modulate the functional activity of PM Ca 2+ channels

73 Most individual studies were small, less than 50 participants, but there was not much heterogeneity

Some experimental studies have reported that UA stimulates the synthesis of proinflammatory cytokines such as IL-1 beta, IL-6, IL-8, and TNF-alpha