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in vitro experiments demonstrated that SGK1 knockdown attenuated erastin-induced downregulation of Slc7a11, GPX4, GSH, and oxidized glutathione (GSSG), while reducing lipid peroxidation, iron accumulation, and mitochondrial damage upregulation, suggesting SGK1's critical role in CHD-associated ferroptosis (44)

Of note, the results of these additional five groups in all the measured parameters and the histopathological findings were non-significant, compared to those of the normal control group

TB-500 shows systemic distribution characteristics enabling whole-body effects[19]

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