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H 2 O 2 is an efficient activator of the redox-regulated proinflammatory transcription factor NF-KB, where redox-regulation activates upstream kinases P13K and NIK leading to NF-KB translocation and promotor binding

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Heavy metals, such as lead, mercury, and cadmium, can deplete glutathione levels, impairing the body's ability to detoxify harmful substances and reducing the availability of SAMe for methylation

For SAH, the endovascular perforation model is widely used because it can simulate SAH caused by rupture of aneurysm ( Degradation products of hemoglobin (ferrous iron and heme) play an essential initiating role in the induction of these pathophysiological events

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