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Individual peptides vary in the amount of available research, and results differ from person to person
1 Introduction Sepsis-associated encephalopathy (SAE) represents a severe complication of sepsis, defined by diffuse brain dysfunction without direct central nervous system (CNS) infection ( Oxidative stress is central to SAE pathogenesis, driven by an imbalance between excessive reactive oxygen species (ROS) production and impaired antioxidant defense systems ( The antioxidant defense system, especially GSH metabolism, is linked to neurodegenerative diseases ( Proteomics and metabolomics enable systematic profiling of protein expression and metabolic signatures
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A.et al (2023b)
Given that previous studies have implicated transcriptional CDKs (e.g., CDK7, CDK9, CDK12/13) in SF3B1 phosphorylation 75,76,77,78,79 , we compared the effects of their inhibition with OTS964
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