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In conclusion, the VCPIP1-AMPK1 axis mediates non-canonical regulation of cardiomyocyte AMPK-T172 phosphorylation

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protein misfolding, mislocalization of TDP-43 to the cytoplasm, ER stress, inhibition of ER-Golgi transport, and apoptosis, in neuronal cells expressing pathological forms of TDP-43 or SOD1 (Parakh et al., 2020)

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