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doi: 10.1016/j.febslet.2004.07.024 150 YaoDBrownleeM

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Data suggests the peptide modulates the release of adrenocorticotropic hormone (ACTH) and limits baseline cortisol levels during simulated stress events inside cell cultures

That fragment acts on a target the same Washington State group helped pin down: the so-called AT4 receptor, which was identified as IRAP, insulin-regulated aminopeptidase, an enzyme rather than a classical receptor (Albiston et al., J Biol Chem 2001)