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The GLP-1 RA class has grown in the last decade with several agents available for use in the US and Europe and several more in development

This may be mediated mainly by activating proopiomelanocortin neurons, which, in turn, activate hypothalamic melanocortin 4 receptors (MC4R) that receive projections of POMC fibers, responsible for the anorexigenic activity [130,131]

These studies reveal that GLP-1 RAs not only decrease oxidative stress and inflammation in endothelial cells but also inhibit cardiac hypertrophy and fibrosis by regulating myocardial proliferation and apoptosis [76]