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231 Loss of PINK1-Parkin exacerbates KRAS-driven pancreatic tumorigenesis, 230 whereas PINK1 acts as a tumor suppressor in glioblastoma by reducing ROS and HIF activation

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Farrell GC, Wong VW, Chitturi S

With the advancement of research, the understanding of COPD etiology has expanded from exposure to harmful gases/particles to multiple etiological types. New classifications of COPD have emerged, including the pulmonary cachexia phenotype, COPD-bronchiectasis overlap phenotype, rapid progressive phenotype, non-smoking COPD phenotype, comorbidity/systemic phenotype, upper lobe-predominant emphysema phenotype, 1-antitrypsin deficiency phenotype, and early disease-related phenotypes (pre-COPD, early-onset COPD, preserved ratio impaired lung function) ( In contrast, an endotype represents the underlying biological or molecular mechanisms that produce these observable traits