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It has been described unfavorable cardiovascular effects probably related to the blockade of ischemic preconditioning mentioned before, an endogenous mechanism that protects the heart from ischemic injury associated with hypoglycemic episodes producing electrocardiographic abnormalities and arrhythmias (Marques et al

6o) which inhibits the ATP synthase, and thus, suggests that ADP phosphorylation capacity is unchanged

Moreover, miRNAs like miR-19b-3p have shown the ability to induce ferroptosis in osteoarthritic chondrocytes by targeting SLC7A11 [92]

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GHK-CU is a copper(II)-complexed tripeptide (molecular weight: ~401 g/mol) consisting of glycine-histidine-lysine chelated to copper ions, naturally present in human plasma