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Lobeline does not impact SG dynamics through alterations in mTOR signaling or inhibition of autophagy Previous work has demonstrated a down regulation of mammalian target of rapamycin (mTOR) signaling during retention of hypoxia-induced SGs [18, 34]

1 Introduction Multiple Sclerosis (MS) is a chronic, immune-mediated, demyelinating disease of the central nervous system (CNS) that affects 2.8 million people world-wide (1)

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