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The following dosing information is compiled from individual compound clinical trial data, peptide research community reports, and the pharmacological profiles of each compound

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At the same time these astrocytes reduce expression of several key homoeostatic proteins such as glutamate transporters, glutamine synthetase, K ir 4.1 inward rectifying channels involved in K + buffering, and connexion 43 responsible for syncytial coupling

doi: 10.1007/s00535-013-0862-6 51 GargusMNiuCValloneJGBinkleyJRubinDCShakerAet al