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Therefore, future research should focus on deepening our understanding of ferroptosis mechanisms in CAD and developing more effective and safer clinical intervention strategies to enhance CHD management

Brefeldin A (BFA), a ER-to-Golgi transport inhibitor, and monensin, a Golgi-to-PM transport inhibitor, abrogated VRK2 overexpression induced increase in the expression of SLC7A11 at PM level (Fig

Higher concentrations of glycochenodeoxycholic acid, glycodeoxycholic acid, hyodeoxycholic acid, and certain bile acid ratios were associated with patient CSF tau and p-tau