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Taken together, docking analyses suggest that neuromedin C engages GLP-1R through an interaction architecture resembling that of GLP1 and other peptide agonists, while remaining fundamentally distinct from the compact TM-centered binding patterns typical of non-peptidic ligands
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Bile acids in IBD suppress NF-B via FXR in monocytes, reducing cytokine storms
Both reactions of GSH synthesis require ATP
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