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The destruction of neurovascular units in Alzheimers disease is related to the increase in the production of adhesion molecules called VCAM-1 [31]

The addition of glucagon receptor agonism introduces some theoretical safety considerations not seen with GLP-1 agonists alone

Based on the molecular weight, isolated LPS can potentially cross the epithelium through the paracellular pathway as has been suggested in some studies (18)

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