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Besides, it was proved that the proliferation, lipid metabolism, and immune evasion of CC cells with PRKN overexpression could be partially remedied by SREBF2 overexpression and strengthened by ACAT2 knockdown (Fig

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36 days), a larger number of infants free of neonatal complications (17 (85%) vs

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Immunoreactive alpha-interferon in insulin-secreting beta cells in type 1 diabetes mellitus