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Insufficient GSH glutathione in the human body will produce a lot of harmful free radicals, endangering the health of the body

Abstract Prior studies have shown that inhibiting ferroptosis could facilitate neural repair following traumatic spinal cord injury (TSCI), yet the underlying mechanisms remain unclear

Owing to their distinct biochemical mechanisms that suppress lipid peroxidation and reduce ROS production, these compounds have been investigated in clinical studies to assess the therapeutic value of their antioxidant properties (Table 1)

International journal of health sciences, 9(4), 468474

In mitochondria, GSH regulates apoptosis via modulation of oxidative phosphorylation and cytochrome c activity