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Adding exogenous glutathione to a drug that already modulates oxidative stress raises a conceptual question: could the combined antioxidant load suppress reactive oxygen species (ROS) to a degree that interferes with beneficial ROS signaling

Reducing human exposure to aflatoxins through enterosorption

Importantly, decreased Y198- and Y250-nitration was visualized by re-probing the blots with 3A11C6 anti-nitroY198-HGF and 6B82C3 anti-nitroY250-HGF-specific mAbs, respectively (Figure 1b,c, first rows)

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